E-mail: karin.vargova@gmail.com
Odborné práce publikované na Linkos.cz
Přehled odborných textů publikovaných autorem na portálu Linkos.cz. Jsou zde uvedeny knihy a brožury, články v Klinické onkologii, tuzemská abstrakta z databáze abstrakt a další texty. Rejstřík není v žádném případě úplným autorským rejstříkem, protože jsou zde uvedeny pouze texty zveřejněné na portálu Linkos.cz.
Publikovaná abstrakta
- 5-Azacytidine and G-CSF Derepressed Chromatin Structure of PU.1 and Its Targets Cebpa and Cbfb In Myelodysplastic Syndrome (MDS)
- Activation of Chromatin Structure Upstream PU.1 Gene and in Vitro Differentiation in High Risk Myelodysplastic Syndrome Following 5-Azacytidine
- Active Chromatin Structure near MYB Occupancy at the Mir-155 Host Gene Promoter Coincides with Increased Mir-155 and MYB Levels In Chronic Lymphocytic Leukemia
- Disruption of a Functional Relationship Between PU.1 and Mir-155 during the Pathogenesis of Chronic Lymphocytic Leukemia (CLL)
- Gata1 Regulates Erythroid Transcription by Cooperating with Chromatin Remodeling Protein Snf2h
- ISWI ATPase Snf2h Is Required for Both Heterochromatin and Euchromatin Structure in ES Cells.
- MicroRNA Mir-155 and Myb Proto-Oncogene Family Members Cooperate in Pathogenesis of Chronic Lymphocytic Leukemia
- MikroRNA polycistrony miR-17-92, miR-106b-25 and miR-106a-363 asociované s lymfoidním vývojem jsou přímé cíle transkripčního faktoru PU.1
- Mutual Regulatory Loop between miR-155 and PU.1 Is a Candidate Pathogenesis Factor in CLL.
- Porušení rovnováhy vzájemné regulace mezi PU.1 a miR-155 je kandidátním mechanizmem leukemogeneze CLL
- PU.1 and p53 Double Mutant Mice Develop Aggressive AML with Dysplastic Features
- Retroviral rescue with PU.1 induces derepression of both PU.1 and miR155 target genes - possible therapeutic implications for CLL
- Snf2h regulates heterochromation structure and global transcription in blood cells
- The Oncogenic Mir-17-92 MicroRNA Cluster Is Inhibited by EGR2 During Macrophage Differentiation Via JARID1b-Mediated Histone 3 Lysine 4 Demethylation
- Tracking the Somatic Mutations in Azacitidine-Treated MDS Patients Documents Clonal Development and AZA Responsiveness
- Transcription Factor CTCF Inhibits Effects of 5-Azacitidine in MDS/AML Cells
- Transcription Factors PU.1 and EGR2 Inhibits the Oncogenic Microrna Cluster Mir-17-92 during Macrophage Differentiation
